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23,49 This severe dysbiosis leads to the collapse of gut barrier function, allowing bacteria and their products (such as LPS and ammonia) to translocate to the portal vein and systemic circulation, directly driving the occurrence of fatal complications such as HE and spontaneous bacterial peritonitis (SBP)

These single-cell expression profiles systematically delineate the spatiotemporal regulation patterns of ferroptosis-suppressing signature genes within the renal disease microenvironment, providing direct cellular and molecular evidence for elucidating the mechanisms of ferroptosis imbalance in AKD and CKD pathogenesis, while offering important references for subsequent research on intervention strategies targeting ferroptosis-suppressing pathways in kidney diseases

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Szyller et al., 2022)